Susan M. Carlton, Ph.D.

Professor

Affiliations: Department of Neuroscience and Cell Biology

Phone: (409) 772-2124
Fax: 409) 772-2789
Email: smcarlto@utmb.edu

Education

Ph.D. in Anatomy, 1982, Medical College of Virginia
Postdoctoral Fellow UTMB
Postdoctoral Fellow, Yale University
Sabbatical with Professor Martin Koltzenburg, University of Wurzberg, Germany

Research Interests

Our research activities are concerned with analysis of neuroplasticity at the level of the spinal cord and peripheral nerve terminal, with emphasis on elucidating mechanisms underlying chronic pain states. One focus in the laboratory has been glutamate-mediated events in peripheral neuropathy. We are investigating the neuroplasticity that occurs following nerve injury in several animal models of painful peripheral neuropathy. Anatomical, behavioral and electrophysiological techniques are used to understand the role of glutamate receptors in the development and maintenance of sensory abnormalities that arise following various nerve injuries. A second focus is defining the receptors involved in the processing of nociceptive input in the periphery. In these studies, anatomical, behavioral, electrophysiological and pharmacological techniques are used to understand how sensory input is integrated at the level of the peripheral terminal. It is becoming clear that a variety of receptors are present on peripheral axons which, when activated, influence sensory transduction. A current focus is the interaction of somatostatin and TRPV1 receptors on peripheral nociceptors. Investigating neuronal receptor populations in normal skin and changes in these populations in chronic pain states may elucidate new avenues for relief of pain of peripheral origin.

Selected Publications

Carlton, S.M., Zhou S., Du J., Hargett G.L., Ji G., and Coggeshall, R.E., Somatostatin modulates the transient receptor potential vanilloid 1 (TRPV1) ion channel. Pain, 110(3): 616-27, 2004.

Jimenez-Andrade J.M., Zhou S., Du J., Yamani A., Grady J.J., Castaneda-Hernandez G., and Carlton, S.M., Pro-nociceptive role of peripheral galanin in inflammatory pain. Pain, 110(1-2): 10-21, 2004.

Coggeshall, R.E., Tate S., and Carlton, S.M., Differential expression of tetrodotoxin-resistant sodium channels Nav 1.8 and Nav 1.9 in normal and inflamed rats. Neuroscience Letters, 355(1-2): 45-89, 2004.

Coggeshall, R.E. and Carlton, S.M., Control of postganglionic sympathetic efferent fibers by neurokinin 1 receptors in rats. Neuroscience Letters, 353(3): 197-200, 2003.

Carlton, S.M., Zhou S., Kraemer B. and Coggeshall, R.E., A role for peripheral somatostatin receptors in counter-irritation-induced analgesia. Neuroscience, 120(2): 499-508, 2003.

Du, J., Zhou, S., Coggeshall, R.E. and Carlton, S.M., N-methyl-D-aspartate (NMDA)-induced excitation and sensitization of normal and inflamed nociceptors. Neuroscience, 118(2): 547-62, 2003.

Carlton, S.M. Localization of CamKIIa in rat primary sensory neurons: increase in inflammation. Brain Research, 947: 252-259, 2002.

Carlton, S.M. and Coggeshall, R.E., Inflammation-Induced Up-Regulation of NK1 Receptors in Rat Glabrous Skin. Neuroscience Letters, 326: 29-32, 2002.

Neugebauer, V. and Carlton, S.M., Peripheral metabotropic glutamate receptors as drug targets for pain relief. Expert Opinion, 6(3): 349-361, 2002.

Carlton, S.M. and Hargett, G.L., Stereological analysis of CaMKII alpha-containing dorsal root ganglion cells in the rat: Co-localization with I-B4, CGRP or VR1. J. Comp. Neurol., 448: 102-110, 2002. .

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