Jin Mo Chung, Ph.D.,
Professor
- Affiliations:
Department of Neuroscience & Cell Biology
- Route: 1069 2.102 Medical Research Building (MRB)
- Tel: (409) 772-2106
- Fax: (409) 772-4687
- jmchung@utmb.edu
Jin Mo Chung, Ph.D.
Education
Dr. Chung is a native of Korea. He received a B.S. degree in physics in 1967 from Sogang University in Seoul. In 1972, after completing his military duty as an officer in the Korean Air Force, he came to the United States to do graduate work in the Department of Physiology at Loyola University in Chicago, where he studied the anatomy and physiology of the autonomic nervous system under Dr. R.D. Wurster. He earned his Ph.D. in 1977.
Dr. Chung first came to UTMB in 1977 for postdoctoral study with Dr. Willis. In 1979, he returned to Korea to take a position as Assistant Professor of Physiology in the College of Medicine of Yonsei University in Seoul. He returned to UTMB in 1981 and rose to Professor with tenure in 1990.
About the Lab
Dr. Chung's main research interests include the pathophysiological mechanisms of neuropathic pain and neurobiological mechanisms underlying acupuncture analgesia. Recent findings from Dr. Chung’s lab indicate that reactive oxygen species (ROS), the primary source of cellular oxidative stress, act as critical cellular signaling molecules in the spinal cord mediating various chronic pain. Present efforts are designed to unveil molecular mechanisms on how oxidative stress in the spinal cord is involved in generation and maintenance of chronic pain. (for further information: http://www2.utmb.edu/chunglab)
Selected Publications
Kim, H.K., Kim, J.H., Gao, X., Zhou, J., Lee, I., Chung, K. and Chung, J.M. Analgesic effect of vitamin E is mediated by reducing central sensitization in neuropathic pain. Pain, 122:53-62, 2006.
Kim, H.K., Schattschneider, J., Lee, I., Chung, K., Baron, R. and Chung, J.M. Prolonged maintenance of capsaicin-induced hyperalgesia by brief daily vibration stimuli. Pain, 129:93-101, 2007.
Gao, X., Kim, H.K., Chung, J.M. and Chung, K. Reactive oxygen species (ROS) are involved in enhancement of NMDA-receptor phosphorylation in animal models of pain. Pain, 131: 262-271, 2007.
Lee, I., Kim, H.K., Kim, J.H., Chung, K. and Chung, J.M. The role of reactive oxygen species in capsaicin-induced mechanical hyperalgesia and in the activities of dorsal horn neurons. Pain, 133: 9-17, 2007.
Koo, S.T., Lim, K.S., Chung, K, Ju, Hyunsu, and Chung, J.M. Electroacupuncture-induced analgesia in a rat model of ankle sprain pain is mediated by spinal α-adrenoceptors. Pain, 135: 11-19, 2008.