Jin Mo Chung, Ph.D.
Professor and Interim Chair

  • Affiliations:
    Department of Neuroscience & Cell Biology
  • Route: 1069 2.102 Medical Research Building (MRB)
  • Tel: (409) 772-6708
  • Fax: (409) 772-6735
  • jmchung@utmb.edu
  • Chung CV


Jin Mo Chung, Ph.D.


photoDr. Chung is a native of Korea. He received a B.S. degree in physics in 1967 from Sogang University in Seoul. In 1972, after completing his military duty as an officer in the Korean Air Force, he came to the United States to do graduate work in the Department of Physiology at Loyola University in Chicago, where he studied the anatomy and physiology of the autonomic nervous system under Dr. R.D. Wurster. He earned his Ph.D. in 1977.

Dr. Chung first came to UTMB in 1977 for postdoctoral study with Dr. Willis. In 1979, he returned to Korea to take a position as Assistant Professor of Physiology in the College of Medicine of Yonsei University in Seoul. He returned to UTMB in 1981 and rose to Professor with tenure in 1990.

About the Lab

Dr. Chung's main research interests include the pathophysiological mechanisms of neuropathic pain and neurobiological mechanisms underlying acupuncture analgesia. Recent findings from Dr. Chung’s lab indicate that reactive oxygen species (ROS), the primary source of cellular oxidative stress, act as critical cellular signaling molecules in the spinal cord mediating various chronic pain. Present efforts are designed to unveil molecular mechanisms on how oxidative stress in the spinal cord is involved in generation and maintenance of chronic pain.

Selected Publications

Gao, X., Kim, H.K., Chung, J.M. and Chung, K. Reactive oxygen species (ROS) are involved in enhancement of NMDA-receptor phosphorylation in animal models of pain, Pain, 131: 262-271, 2007.

Lee, I., Kim, H.K., Kim, J.H., Chung, K. and Chung, J.M. The role of reactive oxygen species in capsaicin-induced mechanical hyperalgesia and in the activities of dorsal horn neurons, Pain, 133: 9-17, 2007.

Schwartz, E.S., Lee, I., Chung, K. and Chung, J.M. Oxidative stress in the spinal cord is an important contributor in capsaicin-induced mechanical secondary hyperalgesia in mice, Pain, 138: 514-524, 2008.

Schwartz, E.S., Kim, H.Y., Wang, J., Lee, I., Klann, E., Chung, J.M. and Chung, K. Persistent pain is dependent on spinal mitochondrial antioxidant levels. J. Neurosci. 29: 159-168, 2009.

Lee, K.Y., Chung, K. and Chung, J.M. The involvement of reactive oxygen species in long-term potentiation in the spinal cord dorsal horn. J. Neurophysiol. 103:382-391, 2010.