Ahmed El-Sayed Ahmed, Ph.D.
Professor, Departments of Pathology, Pharmacology and Toxicology, Preventive Medicine and Community Health
University of Texas Medical Branch
301 University Boulevard
Galveston, TX 77555-0609
Office: (409) 772-2877
Fax: (409) 747-1763
|Degree||Institution||Field of Study||Graduation Year|
|B. Pharm. & Pharm. Sci.||College of Pharmacy, Cairo University, Cairo, Egypt||Pharmacology||1966|
|Ph.D.||College of Pharmacy, University of Minnesota, Minneapolis, MN||Medicinal Chemistry||1975|
|Post-Doctoral Fellow||College of Medicine, University of Minnesota, Mpls, MN||1975|
|Summer Fellow||Massachusetts Institute of Technology, Boston, MA||1978|
|Swedish Medical Research Council Fellow||Department of Toxicology, University of Uppsala, Uppsala, Sweden||1982-1983|
|-||Honorary Professor, Cairo University, Cairo, Egypt|
|1964||Achievement Award of Pharmaceutical Sciences, Egypt|
|1971-1973||Melondy Fellowship Award, University of Minnesota|
|1975||NIH Postdoctoral Fellowship Award, University of Minnesota|
|1975-1977||NIH, National Research Service Award|
|1982-1983||Swedish Medical Research Council Fellowship Award|
|2007-2009||Member at Large ; Board of Directors; Association of Muslim Social Scientists|
In the past three decades my colleagues and I focused our research efforts on the impact of chemicals in the environment on human health and its mechanisms. We received NIH grants, conducted research and published the results of our investigations in the appropriate journals. We wrote review and book chapters to express our opinion and current understandings in the field environmental toxicology as related to current and preceding findings in our laboratory. Recent epidemiologic studies indicated that chronic exposure to drinking water disinfectant byproducts (DBP) induce adverse pregnancy outcome including intra uterine fetal growth restriction (IUGR). Lately we investigated the effect of chemicals, such as DBPs, at environmentally relevant levels on fetal development including IUGR.
We acquired, in our laboratory, several preliminary evidences that support the initiation of IUGR in animal models exposed to DPBs. Furthermore, we identified that oxidative stress and chemically-induced reactive oxygen species play a major role in the mechanisms of such adverse activities.
Recent clinical evidences (Free Radicals Research, 41,870-873, 2007), have supported our theories and hypothesis and have detected that oxidative stress (induced environmentally and other wise) in uterine environment is the major cause of IUGR in pregnant women.