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Netanya Sandler Utay, M.D.
Assistant Professor, Division of Infectious Diseases

Netanya Sandler Utay, M.D., Assistant Professor

Netanya Sandler Utay, M.D.
Assistant Professor
Department of Internal Medicine
Division of Infectious Diseases

University of Texas Medical Branch
301 University Blvd, Marvin Graves Bldg., Rm 4.202
Galveston, Texas 77555-0435
Phone: 409.747.0240
Fax: 409.772.6527
Email: neutay@utmb.edu


Degree/Training Completed Year Name & Location
M.D. 2003 Baylor College of Medicine, Houston, Texas
Internal Medicine 2006 University of Washington, Seattle, Washington
Infectious Diseases      2013 Vaccine Research Center, NIAID, NIH, Bethesda, MD


Dr. Utay joined UTMB in 2013 as an Assistant Professor in the Department of Internal Medicine in the Division of Infectious Diseases. She has an active research program related to the immunopathogenesis of viral infections with a specific focus on HIV and hepatitis C virus. Dr. Utay's interest in infectious diseases started during the year she spent in the Laboratory of Parasitic Diseases at the NIH as a Howard Hughes Medical Institute-National Institutes of Health Research Scholar. After returning to medical school at Baylor, she proceeded to internal medicine residency at the University of Washington and infectious diseases fellowship at the NIH, where her interest in immune activation in HIV and hepatitis C infections started. She co-chairs two clinical trials with the AIDS Clinical Trials Group (ACTG) and serves on the End Organ Disease and Inflammation Transformative Science Group and Inflammation Think Tank of the ACTG. Dr. Utay is interested in the role of gut damage, immune activation, and fibrosis in disease progression in these viral infections and their sequelae, including cardiovascular disease, malignancy, and other end-organ diseases. She is active in the clinical practice of infectious diseases including an HIV clinic and in the teaching of medical students, residents, and fellows.

Research Interests

  • Immunopathogenesis of HIV and HCV infection
  • Residual end-organ disease after treatment for HIV or HCV
  • Role of microbial translocation and intestinal damage in chronic inflammation
  • Interventional studies to attenuate immune activation and microbial translocation in HIV infection.

Select Publications

» PubMed

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